Tag: Kidney function

For patients with healthy kidney function, approximately 90% of a gadolinium-based contrast agent (GBCA) is eliminated from the body within 24 hours. This natural process answers the question of how to get rid of gadolinium after an MRI for most people, but some trace amounts can be retained long-term.

In patients with normal renal function, gadolinium-based contrast agents (GBCAs) have a biological half-life of about 1.5 to 2 hours [1.2.1, 1.3.3]. This article explains in detail how long it take for gadolinium to leave the body and what factors influence its retention.

According to a meta-analysis, all nonsteroidal anti-inflammatory drugs (NSAIDs) can have modest effects on blood pressure. This happens because naproxen, like other NSAIDs, can reduce blood flow to certain organs, particularly the kidneys, by inhibiting specific compounds. Understanding this mechanism is crucial for assessing how **can naproxen reduce blood flow?** and its overall cardiovascular impact.

The human kidneys produce erythropoietin, a vital hormone that regulates red blood cell production. But what is erythropoietin also known as? The most common short-hand for this hormone is EPO, a term widely used in both medical and athletic contexts.

While most people tolerate ibuprofen well, a 1975 case study published in PubMed documented a 15-kg weight gain in a patient during the third week of ibuprofen therapy due to edema. This shows that, in some cases, ibuprofen can indeed cause fluid retention and weight gain.

Yes, nonsteroidal anti-inflammatory drugs (NSAIDs) like ibuprofen can cause edema by inhibiting prostaglandins, which help regulate kidney function. In fact, cases of significant weight gain due to edema have been documented during ibuprofen therapy. So, can ibuprofen cause edema? The answer is a definitive yes, and understanding the mechanism is crucial for managing this potential side effect.

Yes, ibuprofen can cause puffiness as a potential side effect due to fluid retention, also known as edema. This reaction is linked to how the nonsteroidal anti-inflammatory drug (NSAID) affects kidney function and the body's management of salt and water.

While it is a common misconception that drugs are primarily eliminated by the kidneys, approximately 90% of lidocaine is metabolized by the liver before being excreted. The question, 'Is lidocaine processed through the kidneys?', has a more complex answer involving both the liver and the renal system.

While designed for localized pain relief, a tiny fraction of topical lidocaine, the active ingredient in Aspercreme with Lidocaine, is absorbed into the bloodstream. This article explores the pharmacological journey of this medication to address a common question: does aspercreme with lidocaine affect the kidneys? (Markdown OK)

Approximately 7% of patients undergoing non-cardiac surgery with general anesthesia develop postoperative acute kidney injury (AKI). Understanding **how does anesthesia affect renal function** involves examining complex hemodynamic, hormonal, and drug-specific factors.