Tag: Pea

The American Heart Association (AHA) removed routine atropine use from its Advanced Cardiac Life Support (ACLS) protocols for asystole and pulseless electrical activity (PEA) in the 2010 guideline updates, citing a lack of evidence of therapeutic benefit for these specific rhythms.

Phenylethylamine (PEA) and palmitoylethanolamide (PEA) are two completely different substances, and confusing them could have significant consequences, especially when considering what does PEA do to you, as one acts as a natural stimulant while the other modulates inflammation and pain.

First discovered in egg yolks in 1957, palmitoylethanolamide (PEA) is a naturally produced fatty acid amide that has since been studied for its significant anti-inflammatory and analgesic properties. People who take PEA often wonder how the compound influences their overall well-being, both physically and mentally, as it works subtly within the body's endocannabinoid system to regulate sensation and mood.

While some cardiac arrests are treatable with defibrillation, PEA is a non-shockable rhythm, meaning an electrical shock is ineffective and potentially harmful. Therefore, there is no single effective treatment for PEA, but rather a multi-faceted approach focused on high-quality CPR, medication, and identifying the underlying cause.

According to clinical studies involving thousands of patients, palmitoylethanolamide (PEA) is generally considered a safe and well-tolerated supplement, often with side effects comparable to a placebo. Despite its favorable safety profile, understanding **who should not take palmitoylethanolamide** is essential, as certain populations require extra caution or should avoid its use due to insufficient safety data or potential risks.

Palmitoylethanolamide (PEA) is a naturally occurring fatty acid amide produced in the body and found in foods like eggs and peanuts. Research indicates it is a generally safe anti-neuroinflammatory dietary supplement, with most clinical trials reporting a favorable tolerability profile. While considered low-risk, understanding its specific safety aspects is crucial for anyone considering its use.

First isolated from egg yolk in the 1950s, palmitoylethanolamide (PEA), or palmitic acid monoethanolamide, is an endogenous fatty acid amide that serves as an analgesic and anti-inflammatory agent. It is used to help manage various chronic and neuropathic pain conditions.

Phenylethylamine (PEA) is a naturally occurring neurochemical sometimes called the “love drug” because the brain releases it during the excitement of infatuation. However, this amphetamine-like compound's role in romance is often exaggerated, and its effects on the brain are complicated by rapid metabolism.

First isolated from lipid fractions of egg yolks in 1957, palmitoylethanolamide (PEA) has been studied for its potent anti-inflammatory and analgesic properties for decades. Today, many ask, "what is palmitoylethanolamide used for?" as it gains attention as a dietary supplement for various chronic inflammatory and pain conditions.